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. 2005 Jun 20;102(26):9365–9370. doi: 10.1073/pnas.0503777102

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Copyright © 2005, The National Academy of Sciences

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Fig. 2. — Short- and long-term synaptic plasticity in the corticoamygdala pathway of Rab3A-knockout mice. (A) The PPF ratio (fEPSP2/fEPSP1 amplitude) at intervals of 40, 50, 80, and 100 ms was significantly enhanced in the Rab3A-knockout (open squares) compared with the wild-type (filled circles) mice. (Insets) Sample traces from wild-type (+/+, upper trace) and knockout (-/-, lower trace) mice. (B) LTP elicited by 3 × 2 trains (*, 100 Hz, 1s) was significantly reduced in Rab3A^-/- (filled squares) compared with wild-type (open circles) mice. (Insets) Sample traces 10 min before and 30 min after tetanus. (C) LTP induced by a 20-Hz train (45 s) was significantly depressed in Rab3A^-/- (filed circles) compared with wild-type (open circles) mice. (D) LTP elicited by 3 × 2 trains of tetanus (*, 100 Hz, 1 s) in the RIM1α^-/- (filled circles) mice was not significantly different from that in wild-type (open circles) mice. (Scale bars: 10 ms, 1 mV.)