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. 2024 Dec 10;15:1514367. doi: 10.3389/fphar.2024.1514367

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Copyright © 2024 Li, Meng, Yang and Hu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Growth changes of cancer cells were detected by experiments after the NDC1 expression changing. (A) The results of CCK-8 assay indicated that the viability of cancer cells was weakened after inhibition of NDC1. (B) After overexpression of NDC1, the results of CCK-8 assay showed that cancer cell viability was enhanced. (C) After NDC1 knockdown, the results of clone formation experiments showed that the migration ability of cancer cells had reduced. (D) After overexpression of NDC1, the results of Clone formation experiment confirmed that the migration ability of cancer cells was improved. (E) After NDC1 knockdown, the results of transwell assay showed that the invasion ability of cancer cells was weakened. (F) After overexpression of NDC1, the results of transwell assay confirmed that the invasion ability of cancer cells had enhanced. (G) After inhibition of NDC1, the expression of E-cadherin protein in cancer cells was enhanced, and the protein expression of Vimentin was weakened. After overexpression of NDC1, the expression of E-cadherin protein in cancer cells was decreased, and the protein expression of Vimentin was increased. (H) Histogram was used to show the results of E-cadherin and Vimentin in A549 cells.