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. 2024 Sep 16;40(2):491–503. doi: 10.1007/s00467-024-06487-2

Table 5.

Treatment strategies for ABMR episodes (n = 51)

Treatment strategies N (%)
Acute ABMR n = 19
Rituximab, IVIG, optimization, and/or steroid pulse 5 (26.3%)
Rituximab, PF/IA, steroid pulse, w/o IVIG 4 (21.1%)
Eculizumab or rituximab, thymoglobulin, PF/IA, steroid pulse 3 (15.8%)
Thymoglobulin, steroid pulse, PF/IA, or optimization 2 (10.5%)
Steroid pulse, w/o IVIG 2 (10.5%)
Optimization 2 (10.5%)
Bortezomib, rituximab, PF/IA, optimization, steroid pulse, IVIG 1 (5.3%)
Chronic active ABMR n = 18
Rituximab, IVIG, optimization, and/or steroid pulse 5 (27.8%)
Optimization or no treatment 4 (22.2%)
Thymoglobulin, optimization, w/o steroid pulse 2 (11.1%)
Rituximab, PF/IA, optimization, IVIG, w/o steroid pulse 2 (11.1%)
IVIG, steroid pulse, optimization 2 (11.1%)
Bortezomib, steroid pulse, optimization 1 (5.6%)
Tocilizumab, PF/IA, IVIG, optimization 1 (5.6%)
Thymoglobulin, PF/IA, steroid pulse 1 (5.6%)
Mixed ABMR/TCMR n = 14
Rituximab, PF/IA, steroid pulse, IVIG, and/or optimization 5 (35.7%)
Rituximab, steroid pulse, w/o IVIG, w/o optimization 3 (21.4%)
PF/IA, steroid pulse, w/o IVIG and optimization 2 (14.3%)
Eculizumab or thymoglobulin, rituximab, PF/IA, steroid pulse, IVIG, w/o optimization 2 (14.3%)
Steroid pulse, w/o IVIG and optimization 2 (14.3%)

ABMR antibody-mediated rejection, IVIG intravenous immunoglobulin G, PF plasmapheresis, IA immunoadsorption, Optimization increase or change of maintenance immunosuppression, w/o with or without