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Schematic summary of the effect and mechanism of baicalin on H2O2-induced oxidative stress, inflammation and apoptosis in BMECs. Baicalin resists H2O2-induced oxidative damage in BMECs by activating the Keap1/Nrf2 signaling pathway and reducing ROS production, which in turn inhibits NF-κB inflammation and the caspase3/Bcl-2 apoptotic pathway to maintain intracellular redox homeostasis.
