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. 2024 Dec 28;14:30663. doi: 10.1038/s41598-024-75251-1

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — Correlation analysis between diagnostic markers and infiltration-related immune cells and construction of diagnostic markers-targeted drugs network. (A) Correlation analysis between PLAUR and infiltration-related immune cells. (B) Correlation analysis between HMOX1 and infiltration-related immune cells. (C) Scatter diagram indicating the correlation between PLAUR expression and Mast cells activated. The expression of PLAUR was negatively correlated with Mast_cells_activated (r = − 0.502, p = 0.034). (D) Scatter diagram indicating the correlation between HMOX1 expression and Neutrophils. The expression of HMOX1 was positively correlated with Neutrophils (r = 0.538, p = 0.021). (E) PLAUR and HMOX1-targeted drugs network. Red represented diagnostic markers and light blue represented the drugs. (F) Molecular docking of diagnostic markers- targeted drugs. The binding energies of HMOX1-STANNSOPORFIN is − 2.51 kcal/mol.