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Exerkines are released into the bloodstream through exercise, influencing cardiac vessels and cardiomyocytes. They enhance mitochondrial function, promote vascular relaxation, and contribute to antimyocardial hypertrophy effects. Increased NO activates GC to catalyze the synthesis of GTP into cGMP, promoting vascular relaxation, and the balance between TXA2 and prostacyclin also contributes to this effect.
