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. 2024 Dec 28;14:31396. doi: 10.1038/s41598-024-82994-4

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 4 — The independent correlation of MAR with MASLD in different Models. Model 1: adjustment for age, gender, diabetic duration, and drinking. Model 2: further adjustment for waist circumference, body mass index, systolic blood pressure, diastolic blood pressure, hemoglobin A1c, triglycerides, high-density lipoprotein, homeostasis model assessment of insulin resistance, and uric acid based on Model 1. Model 3: additional adjustment for alanine aminotransferase and aspartate aminotransferase, as well as the usage of hypoglycemic agents like metformin, thiazolidinediones, sodium-glucose cotransporter-2 inhibitors, and glucagon-like peptide-1 receptor agonists based on Model 2. MASLD Metabolic dysfunction-associated fatty liver disease, MAR Monocytes to Apolipoprotein A1 ratio.