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. 2025 Jan 7;31(1):101198. doi: 10.3748/wjg.v31.i1.101198

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©The Author(s) 2025. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: https://creativecommons.org/Licenses/by-nc/4.0/

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ASH1L promotes an immunosuppressive microenvironment in hepatocellular carcinoma. A: Volcano plot of differentially expressed genes between high and low ASH1L groups; B: Kyoto encyclopedia of genes and genomes; C: Gene ontology pathway enrichment between high and low ASH1L groups; D: Correlation between ASH1L expression and 28 immune cell infiltration in the cancer genome atlas-liver hepatocellular carcinoma; E: Correlation between ASH1L expression and 28 immune cell infiltration in international cancer genome consortium, GSE76427 and GSE14520. MDSC: Myeloid-derived suppressor cell; GO: Gene ontology; KEGG: Kyoto encyclopedia of genes and genomes; CD: Cluster of differentiation; ICGC: International cancer genome consortium.