FIG. 1.

Motility phenotype of different P. aeruginosa strains used in this study. Panel A shows the motile wild-type P. aeruginosa strains PAK and PAO1 and mutant strains that were defective in motility because of mutations in the fliC gene (PAKΔC and PAOC⁻) or motABCD genes (PAKmotAB). Panel B shows the glycosylation mutant strains PAKrfbC and PAOrfbC, which were both motile. Panel C shows the complemented strains in which fliC mutant strain PAKΔC was chromosomally complemented with different flagellins with their promoters.