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. 2024 Dec 30;15:10736. doi: 10.1038/s41467-024-55038-8

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 1 — A Flow chart of sample collection and study design. B Volcano plot depicting the differentially expressed proteins between patients with AD (n = 30) and matched healthy counterparts (n = 30). C Gene set enrichment analysis illustrating the hallmark pathways enriched in the patients with AD, including leukocyte activation and chemotaxis, neutrophil migration, and inflammatory process. D GO enrichment analysis showing enrichment of protein-DNA subunit organization and complex assembly, nucleosome organization and assembly in peripheral blood from patients with AD. E KEGG analysis showcasing enrichment pathways related to neutrophil extracellular traps (NETs) formation, platelet activation and actin regulation in peripheral blood from patients with AD. A two-sided statistical significance was set at adjusted p < 0.05. AD: aortic dissection; GO: Gene ontology; KEGG: Kyoto encyclopedia of genes and genomes.