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. 2024 Dec 20;18:1520710. doi: 10.3389/fnins.2024.1520710

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Copyright © 2024 Yao, Hu, Li, Wang, Pan and Fan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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EE protects against brain damage after an ischemic stroke. (A) The time of latency to fall in the rota-rod test during the 2-day training (n = 18). (B) The brain damage was evaluated by mNSS scores (n = 12). (C) The T2-weighted images of whole-brain MRI on day 3 after the PT stroke showed the infarct area (increased signal intensity). (D) 2,3,5-Triphenyltetrazolium chloride (TTC) staining of brain slices 3 days after PT stroke showed the infarct area (white). (E) The relative proportion and quantification of infarct volume by TTC staining (n = 3). ns: no significant, *p < 0.05, ***p < 0.001, ****p < 0.0001. Error bars were represented as mean ± SD.