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. 2024 Dec 20;18:1520710. doi: 10.3389/fnins.2024.1520710

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Copyright © 2024 Yao, Hu, Li, Wang, Pan and Fan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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EE upregulated the expression of TREM2 in the hippocampus after surgery in mice with ischemic stroke. (A,B) The expression of TREM2 protein in the hippocampus on day 10 after surgery (n = 3). (C,D) The expression of DAP12 protein in the hippocampus on day 10 after surgery (n = 3). (E) The expression of TREM2 mRNA in the hippocampus on day 10 after surgery (n = 3). (F) The expression of DAP12 mRNA in the hippocampus on day 10 after surgery (n = 3). (G) The immunofluorescence of the cellular localization of TREM2 in the hippocampus of mice in the Sham+EE group on day 10 after surgery (n = 3). DG: dentate gyrus. Scale bars represented 10 μm and 50 μm. ns: no significant; *p < 0.05, ***p < 0.001, the Sham+SE group vs. the PT + SE group; #p < 0.05, ##p < 0.01, the PT + SE group vs. the PT + EE group; ^p < 0.05, ^^^p < 0.001, the Sham+SE group vs. the Sham+EE group. Error bars were represented as mean ± SD.