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. 2024 Dec 20;18:1520710. doi: 10.3389/fnins.2024.1520710

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Copyright © 2024 Yao, Hu, Li, Wang, Pan and Fan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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EE activated the PI3K/Akt signaling pathway in the hippocampus after surgery in mice with ischemic stroke. (A) The results of the western blot of p-PI3K and PI3K. (B) The protein p-PI3K / PI3K ratio in the hippocampus on day 10 after surgery (n = 3). (C) Western bolt results of p-AKT and AKT. (D) The protein p-AKT/ AKT ratio in the hippocampus on day 10 after surgery (n = 3). *p < 0.05, **p < 0.01, the Sham+SE group vs. the PT + SE group; #p < 0.05, ####p < 0.0001, the PT + SE group vs. the PT + EE group; ^^p < 0.01, the Sham+SE group vs. the Sham+EE group. Error bars were represented as mean ± SD.