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. 2025 Jan 7;40(1):91. doi: 10.1007/s11011-024-01502-9

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© The Author(s) 2025

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 7 — Involvement of the signalling pathway of RhoA-ROCK2 in the EGb761 mediated proliferative and migratory responses of OGD/R-induced HT22 cells. A−C The expression of PCNA and Bcl-2/Bax in the control, OGD/R, EGb761, and EGb761 + Fasu groups. D−E Cell proliferation of OGD/R-induced HT22 cells in the control, OGD/R, EGb761, and EGb761 + Fasu groups, assessed by EdU assay. Bar = 50 μm. F−G Cell migration of OGD/R-induced HT22 cells in the control, OGD/R, EGb761, and EGb761 + Fasu groups, evaluated by the wound healing assay. Bar = 200 μm. (n = 3) ^**P < 0.01 vs. Control group, ^#P < 0.05, ^##P < 0.01 vs. OGD/R group. ^ΔΔP < 0.01 vs. EGb761 group