Skip to main content
Frontiers in Pharmacology logoLink to Frontiers in Pharmacology
. 2025 Jan 23;15:1504076. doi: 10.3389/fphar.2024.1504076

Corrigendum: D-dencichine regulates thrombopoiesis by promoting megakaryocyte adhesion, migration and proplatelet formation

Shilan Ding 1,2,3,4,, Min Wang 1,2,3,4,, Song Fang 5, Huibo Xu 6, Huiting Fan 7, Yu Tian 1,2,3,4, Yadong Zhai 1,2,3,4, Shan Lu 1,2,3,4, Xin Qi 7, Fei Wei 1,2,3,4, Guibo Sun 1,2,3,4,*, Xiaobo Sun 1,2,3,4,*
PMCID: PMC11798812  PMID: 39917328

In the published article, there was an error in Figure 7 as published. In Figure 7, the Akt protein bands in the liver group and platelet group were the same in this study. The corrected Figure 7 and its caption, appear below.

FIGURE 7.

FIGURE 7

Thrombopoietin-dependent signaling is enhanced with D-dencichine treatment in mouse liver, platelet, and spleen. (A–C) Western blots analysis of p-ERK1/2/ERK1/2, p-Akt/Akt proteins in liver, platelet, and spleen. Quantification analysis of TPO-dependent signaling proteins from the information in corresponding protein bands. (D) Effects of apoptosis-related protein expression with D-dencichine treatment in mouse platelet. The level of Bcl-2/Bax was determined by western blot. Quantification analysis of Bcl-2, Bax and Bcl-2/Bax expression ratio from the information in corresponding protein bands. ## P < 0.01 vs. control group; * P < 0.05, ** P < 0.01 vs. model group.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.


Articles from Frontiers in Pharmacology are provided here courtesy of Frontiers Media SA

RESOURCES