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. 2025 Mar 6;25:141. doi: 10.1186/s12876-025-03719-z

Table 2.

Clinical characteristics of CLD patients treated at hospitals of Bahir Dar City, 2024 (n = 213)

Variable Category Frequency Percent
Cause of CLD Diagnosed as:
Viral (HBV = 117, HCV = 30) Both HBV & HCV = 6, ALD = 2 141 60.0
HSS ALD = 1, HBV = 3, HCV = 2 43 18.3
ALD HBV = 4, HCV = 1 27 11.5
HMS HCV = 4, HSS = 2 11 4.7
NAFLD 5 2.1
Others TB = 2, Amoeba = 2, Drug = 4, ALD = 1 8 3.4
Complications Ascites 138 64.8
Portal hypertension 114 52.1
Spontaneous bacterial peritonitis 13 6.1
Hepatorenal syndrome 9 4.2
Hepatic encephalopathy 6 2.8
Comorbid condition No 161 75.6
Yes* 52 24.4
Stages of CLD Compensated 114 53.5
Decompensated 99 46.5
End stage liver disease HCC 60 60.6
Cirrhosis 39 39.4
Albumin level ≥ 3.5 g/dl 75 35.2
< 3.5 g/dl 138 64.8
APRI score < 2 130 61.0
≥ 2 83 39.0
FIB-4 Less likely (< 1.45) 59 27.7
Indeterminate (1.45–3.25) 73 34.3
More likely (> 3.25 points) 81 38.0

Took appropriate treatment and regimen

for specific CLD cause

Yes 97 45.5
No 116 54.5
Treatment duration < 1 Years 22 10.3
1–2 Years 119 55.9
> 2 Years 72 33.8
Clinical and serological patient outcome Improved 130 61.0
Died (1 patient was on medication) 5 2.4
Self-discharged (6 were on medication) 13 6.1
No charge (31 took medication) 65 30.5

Key: FIB– fibroscan, HMS - hyper-reactive malarial splenomegaly syndrome, HSS - hepatosplenic schistosomiasis, *Diabetes mellitus [9], heart failure [3], hypertension [14], pneumonia [6], renal disorder [4], tuberculosis [6], venous thromboembolism [3], HIV/AIDS [2] and others; ** no charge (66), self-discharged [13] and death [4]