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. 2025 Apr 16;14(8):600. doi: 10.3390/cells14080600

Table 1.

Proteins involved in the development and progression of PD and MSC-derived factors responsible for the beneficial effects of MSC-EVs in the attenuation of PD.

Molecule(s) Cell Source Effect(s) Ref. No.
Altered LRRK2 kinase Dopaminergic neurons Impaired release of dopamine [3,4]
Altered SNCA protein Dopaminergic neurons Increased aggregation of αSN [3,4]
Altered PARK7 protein Dopaminergic neurons Mitochondrial dysfunction [4,5]
Altered PINK1 kinase Dopaminergic neurons Disruption of mitochondrial homeostasis [4,5]
Altered PRKN ligase Dopaminergic neurons Impaired proteasome-dependent degradation of altered proteins [4,5]
TNF-α, IL-1β Microglia Increased expression of E and P selectins on ECs; increased permeability of BBB [7]
IFN-γ, IL-17 Th1, Th17 cells Activation of microglia and brain-infiltrated neutrophils; progression of neuroinflammation [9]
CD9, CD63, CD81 MSCs Increased uptake of MSC-EVs by target cells [22]
IL-10, TGF-β MSCs Induction of tolerogenic phenotype in DCs; alternative activation of macrophages [22,23]
IL-1Ra MSCs Suppression of IL-1β-driven neuroinflammation [22,23]
IDO MSCs Increased generation and expansion of immunosuppressive Tregs [23]
HGF MSCs Suppression of microglia [23,24]
miR-133b, miR-21, miR-124 MSCs Promote resilience of dopaminergic neurons [23,24]
BDNF, NGF, GDNF MSCs Support growth and survival of dopaminergic neurons [23,24,25]
SOD, catalase MSCs Attenuation of oxidative stress-induced injury of dopaminergic neurons [24,25]
TSG-6 MSCs Suppression of oxidative stress and attenuation of neurotoxicity [37]

Abbreviations: synuclein alpha (SNCA); leucine-rich repeat kinase 2 (LRRK2); parkinsonism associated deglycase (PARK7); α-synuclein (αSN); phosphatase and tensin-homolog (PTEN)-induced kinase 1 (PINK1); parkin RBR E3 ubiquitin protein ligase (PRKN); tumor necrosis factor-alpha (TNF-α); interleukin (IL)-1β; endothelial cell (EC); blood-brain barrier (BBB); interferon gamma (IFN-γ); mesenchymal stem cells (MSCs); mesenchymal stem cell-derived extracellular vesicles (MSC-EVs); transforming growth factor beta (TGF-β); interleukin 1 receptor antagonist (IL-1Ra}; indoleamine 2,3-dioxygenase (IDO); T regulatory cells (Tregs); hepatocyte growth factor (HGF); micro RNA (miR); brain-derived neurotrophic factor (BDNF), nerve growth factor (NGF); glial cell line-derived neurotrophic factor (GDNF); superoxide dismutase (SOD); TNF-stimulated gene-6 (TSG-6).