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. 2025 Jul 17;17(1):2526871. doi: 10.1080/19382014.2025.2526871

Table 1.

SC-islets and human donor islets treated with TCDD ± hypoxia show variability for gene expression changes.

  CYP1A1     HMOX1     AHRR     ARNT     G6PC2    
Cell source TCDD Hypoxia Crosstalk TCDD Hypoxia Crosstalk TCDD Hypoxia Crosstalk TCDD Hypoxia Crosstalk TCDD Hypoxia Crosstalk
SC-Islets ↑ (41-fold) ↑ (1.1-fold) - ↑ (24-fold) x ↑ (6-fold) - x ↑ (1.2-fold) ↑ (1.2-fold) x
R425 - ♂ ↑ (10-fold) - ↑ (2-fold) ↑ (1.3-fold) - - - x - x
R434 -♂ ↑ (3-fold) - x ↑ (1.4-fold) ↑ (5-fold) x ↑ (2-fold) - x ↑ (1.7-fold) ↑ (1.7-fold) x ↑ (2-fold) x
R444 -♂ ↑ (11-fold) - x ↑ (2.5-fold) ↑ (2.6-fold) x ↑ (3-fold) - x ↑ (2-fold) - x ↑ (2-fold) - x
R427 -♀ ↑ (3-fold) x - ↑ (3-fold) x - - x - - x - x
R430 -♀ ↑ (3-fold) - - ↑ (3-fold) x ↑ (1.1-fold) - - - - x
R461 -♀ ↑ (15-fold) ↑ (2-fold) - ↑ (3-fold) x ↑ (2-fold) - x - - x - x

A summary table of the main effects of TCDD (10 nM) ± hypoxia (1% O2) on SC-islets and human donor islets for expression of CYP1A1, HMOX1, AHRR, ARNT, and G6PC2. Bolded fold-increase indicates significance for TCDD or hypoxia treatments alone, compared with vehicle control.