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. Author manuscript; available in PMC: 2025 Aug 6.
Published in final edited form as: Clin Cancer Res. 2025 Sep 2;31(17):3652–3661. doi: 10.1158/1078-0432.CCR-24-3803

Table 1:

Baseline Patient Characteristics

Escalation cohort (n=13) Expansion cohort (n=22) All patients (n=35)
Median age (years) 53 54 54
Race/ethnicity
 Asian 1 (7.5%) 2 (9%) 3 (9%)
 Black 1 (7.5%) 1 (5%) 2 (6%)
 White 7 (54%) 17 (77%) 24 (69%)
 Unknown 4 (31%) 2 (9%) 6 (17%)
 Hispanic 0 1 (5%) 1 (3%)
Prior lines of treatment in the metastatic setting (median) 3 2 2
 Hormone therapy 13 (100%) 22 (100%) 35 (100%)
  Aromatase inhibitors or tamoxifen 13 (100%) 21 (95%) 34 (97%)
  Fulvestrant 9 (69%) 9 (41%) 18 (51%)
  GnRH agonists 3 (23%) 10 (45%) 13 (37%)
 CDK4/6 inhibitor 13 (100%) 22 (100%) 35 (100%)
 PI3K pathway inhibitor 4 (31%) 4 (18%) 8 (23%)
 Chemotherapy 13 (100%) 13 (59%) 23 (66%)
Metastatic Sites
 Bone 13 (100%) 15 (68%) 28 (80%)
 Brain 2 (15%) 0 2 (6%)
 Liver 12 (92%) 8 (36%) 20 (57%)
 Lung 6 (46%) 7 (32%) 13 (37%)
 Lymph node 3 (23%) 6 (27%) 9 (26%)
 Pleural / peritoneal 4 (31%) 1 (5%) 5 (14%)
FGFR gene alteration
FGFR1 amp 9 (69%) 20 (91%) 29 (83%)
FGFR2 amp 0 1 (5%) 1 (3%)
FGFR3 amp 0 1 (5%) 1 (3%)
 No FGFR1-4 amp 4 (31%) 2 (9%) 6 (17%)

Aromatase inhibitors include letrozole, anastrozole and exemestane. GnRH: Gonadotropin-releasing hormone agonists include leuprolide acetate and goserelin acetate.