Figure 1.


IL-12Rβ2-deficient animals are not more susceptible to VSV-induced encephalitis. IL-12Rβ2-deficient (IL-12Rβ2 −/−) and C57BL/6J control mice were infected intranasally with VSV and monitored every other day for morbidity and mortality during the course of VSV pathogenesis. Mortality was monitored daily until no pathology was observed in the surviving animals. These data are the combination of 4 identical, independent experiments.