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. Author manuscript; available in PMC: 2005 Sep 29.
Published in final edited form as: Viral Immunol. 2005;18(2):397–402. doi: 10.1089/vim.2005.18.397

Figure 3.

Figure 3

IL-12Rβ1-deficient animals are not more susceptible to VSV-induced encephalitis. IL-12Rβ1-deficient (IL-12Rβ1 −/−) and C57BL/6J control mice were infected intranasally with VSV and monitored every other day for (A) morbidity and (B) mortality during the course of VSV pathogenesis. (A) Morbidity was monitored by the weight of the animal. The indicated weight is the average weight of all animals monitored (n = 22); error bars represent standard error. Monitoring was continued until the animals showed full recovery from the infection, indicated by a return to pre-infection weights. These data are representative of 4 independent experiments. (B) Mortality was monitored daily until no pathology was observed in the surviving animals. These data are the combination of 4 identical, independent experiments.