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Functional proteasome is required for rtg2Δ bypass phenotype in GRR1-1 and BMH1-1 cells. (A) Mks1p is stabilized by MG132. Mks1p stability was examined in cells as indicated treated with or without MG132 (50 μM). Cells were grown in YNBcasD medium. (B) MG132 inhibits CIT2-lacZ expression in GRR1-1 and BMH1-1 cells. Indicated strains were grown in YNBcasD medium with or without MG132.
