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. Author manuscript; available in PMC: 2025 Aug 25.
Published in final edited form as: Arch Clin Biomed Res. 2025 Jun 30;9(4):304–315. doi: 10.26502/acbr.50170467

Figure 5:

Figure 5:

Effects of TNF-α and EMF exposition on the transcriptional expression of pro-inflammatory M1 phenotypic markers CD86 (A) and CD32 (B) in HMC3 cells. Cells were incubated with TNF-α (50 ng/mL) and exposed to an (EMF) of 2.5 Hz or 5 Hz for 3 min. RNA samples were purified after 6, 24 and 48 h. TNF-α: tumor necrosis factor alpha; EMF: electromagnetic field; CD86: cluster of differentiation 86 or T-lymphocyte activation antigen CD86; CD32: cluster of differentiation 32 or low-affinity immunoglobulin gamma Fc region receptor II phosphatase, receptor type, C (PTPRC).