Table 2.
The influence of different PTMs on phase separation
| PTMs | Targets | Cancer type | Regulation of phase separation | Mechanism | phenotype | Ref |
|---|---|---|---|---|---|---|
| Phosphorylation | HDAC6 | Breast cancer | Promotion | Promote its nuclear entry and chromatin remodeling. | Promote cancer cell proliferation. | [89] |
| RNA Pol II | Cervical cancer, osteosarcoma | Promotion | Promote transcriptional elongation and mRNA processing. | Promote cancer cell proliferation. | [90] | |
| P53 | Cervical cancer | Promotion | Enhance DNA binding activity. | Inhibit tumor initiation and progression. | [91] | |
| FOXM1 | Breast cancer | Inhibition | Reduce chromatin engagement. | Inhibit tumor metastasis, restore tumor immunogenicity. | [92] | |
| CPSF6 | Colorectal cancer | Inhibition | Shorten 3’ UTRs of cell-cycle-related genes to enhance mRNA stability and translation efficiency. | Promote tumor cell proliferation. | [93] | |
| Lactylation | P53 | Cervical cancer, colorectal cancer | Inhibition | Inhibit DNA binding and transcriptional activity. | Promote tumorigenesis. | [94] |
| cGAS | Cervical cancer, osteosarcoma, fibrosarcoma, monocytic leukemia | Inhibition | Inhibit DNA recognition. | Promote immune evasion. | [95] | |
| Methylation | FUS | Cervical cancer, neuroblastoma | Inhibition | Inhibit DNA damage repair. | Promote genomic instability. | [96] |
| Acetylation | RB1CC1 | Breast cancer | Promotion | Inhibit its ubiquitination and degradation. | Promote tumor cell autophagy and growth. | [99] |
| H2A | Cervical cancer, osteosarcoma | Promotion | Inhibit DNA repairing. | Promote genomic instability and tumorigenesis. | [100] | |
| IRF3/IRF7 | Osteosarcoma | Inhibition | Inhibit IFN-I induction. | Promote immune evasion. | [101] | |
| Ubiquitination | PAICS | Melanoma | Promotion | Facilitate constitutive purinosomes formation. | Promote cancer cell proliferation. | [103] |
| PLK4 | Neuroblastoma | Inhibition | Inhibit mitosis. | Inhibit cell proliferation. | [102] | |
| SUMOylation | Telomere-binding proteins | Osteosarcoma | Promotion | Activate the ALT pathway. | Promote replicative immortality of cancer cells. | [104] |
| PARylation | CycT1 | Cervical cancer | Inhibition | Promote DNA repair. | Promote tumorigenesis. | [106] |
| Neddylation | PML/RARα | Leukemia | Inhibition | Enhance DNA-binding ability and inhibit the formation of PML nuclear bodies. | Promote the development and progression of APL. | [107] |