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. 2025 May 23;16(8):e00862. doi: 10.14309/ctg.0000000000000862

Table 1.

Main features of the included study population: n = 84 patients with GD or GC arisen in a histopathological background of MF-AG or CR-AG

All patients MF-AG and CR-AG n = 84 (100%) MF-AG
n = 45 (53.6%)
CR-AG
n = 39 (46.4%)
P
Age at AG diagnosis
 Median, range 70.0, 33–90 70.0, 33–90 70.0, 48-86 0.46
 Mean, SD 69.3, 10.6 70.0, 10.8 68.5, 10.4 0.50
Females, n (%) 54.8 42.2 69.2 0.01
GD-GC diagnosis concomitant to AG diagnosis 35.7 33.3 38.5 0.63
AG diagnosis >1 yr regarding GD-GC diagnosis median, range 4, 1-17 4, 1-12 5, 1-17 0.90
Mean, SD 5.5, 4.0 5.1, 3.2 6.0, 4.8 0.54
Hp eradication treatment before diagnosis of GD-GC 16.7 20.0 12.8 0.30
Positivity toward parietal cell autoantibodies 60.0 51.5 69.7 0.13
First-degree family history for GC 15.2 14.7 15.6 0.92
Active or previous use of proton pump inhibitors 56.4 56.2 56.5 0.98
Active or previous smoking habit 30.4 9/30 (30.0) 30.8 0.95
Type of anemia n = 31
 Iron-deficiency 25.8 30.3 19.4 0.31
 Pernicious 18.8 9.1 29.0 0.04
 Mixed 9.4 12.1 6.5 0.44
 None 46.9 48.5 45.2 0.79

Data expressed as % when not otherwise indicated.

AG, atrophic gastritis, CR, corpus-restricted; GC, gastric cancer; GD, gastric dysplasia; Hp, Helicobacter pylori; MF, multifocal.