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. 2025 Oct 16;13:1675619. doi: 10.3389/fbioe.2025.1675619

TABLE 3.

PBM and JAK/STAT signaling in fibroblasts.

Cells λ (nm) Fluence (J/cm2) Model Statistical analysis Outreads References
WS1 human skin fibroblast cells. Four models, namely, normal (N), wounded (W), diabetic (D) and diabetic wounded (DW) were used 660 5 In vitro Experiments: n = 4, assays in duplicate (average used)
Software: SigmaPlot 13.0
Tests: One-tailed Student’s t-test, One-Way ANOVA
Significance: P < 0.05
Data presentation: Mean ± SEM (with error bars)
- Irradiated diabetic wounded cells showed a significant increase in EGF (p < 0.001) and activation of its receptor (p-EGFR)
- Activation of JAK/STAT pathway (p < 0.001) (p-JAK2, p-STAT1, p-STAT5) was observed and stimulated cell proliferation (p < 0.001), migration (p < 0.05) and viability (p P < 0.001)
- PBM at 660 nm and 5 J/cm2 modulated cellular autocrine signaling, particularly the EGF/EGFR loop
Jere et al. (2018)
WS1 human fibroblasts (wounded and diabetic wounded) 660 5 In vitro Experiments: n = 3
Analysis: RT2 Profiler PCR Array, ΔΔCT method
Normalization: Average of 5 housekeeping genes
Fold change: >1 = upregulation, <1 = downregulation (vs controls)
Significance: P ≤ 0.05
- PBM (600 nm, 5 J/cm2) regulated JAK/STAT pathway genes in both wounded and diabetic wounded WS1 cells
- In wounded cells, 2 genes (INSR and SOCS4) were upregulated
- In diabetic wounded cells, 21 genes were upregulated, including JAK3, TYK2, and STAT1, STAT2, STAT3, STAT5A, STAT6
- The upregulation of these genes activated JAK/STAT signaling, enhancing wound healing through processes like migration and proliferation
Jere et al. (2020)
Human fibroblast cell culture: wounded (W) and diabetic wounded (DW) 830 5 In vitro Experiments: n = 3
Software: Qiagen GeneGlobe
Analysis: ΔΔCT method (2^(ΔΔCT))
Normalization: 5 reference genes (ACTB, B2M, GAPDH, HPRT1, RPLP0)
Significance: p ≤ 0.05
- PBM at 830 nm and 5 J/cm2 altered JAK/STAT pathway genes in both wounded and diabetic wounded cells
- In wounded cells, 5 genes (CEBPB, FCGR1A, GATA3, JUN, SOCS3) were upregulated, and 4 downregulated (CSF1R, ISG15, MYC, SMAD2) - In diabetic wounded cells, 6 genes (EGFR, F2, IL2RG, MPL, MYC, OAS1) were downregulated
Leyane et al. (2024)