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. 2025 Nov 6;25(1):358. doi: 10.1007/s10238-025-01901-x

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© The Author(s) 2025

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 2 — Feature Gene Selection and PPI Network Based on GeneCards, OMIM and TTD. a B[a]P and eosinophils cross-recognized 5,092 targets in COPD. b venn. c PPI network of core targets for DEG and benzopyrene-related genes in COPD and eosinophil cross-genes (node size reflects protein interaction frequency, while edge thickness corresponds to interaction evidence strength. The red-labeled core nodes in the diagram selected via the MCODE algorithm indicate their key regulatory roles within the COPD inflammatory network.