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Monoubiquitination of the Met Y1003F receptor mutant is sufficient to reverse cell transformation. Rat-1 fibroblast cells were infected with retroviruses encoding the various CSF-Met constructs and grown for 3 weeks in the presence of 0.3 nM of colony-stimulating factor 1. (A) Representative pictures of cell monolayers. (B) The graph represents the relative number of foci per dish corrected for the PFU of each retrovirus, from two independent experiments.
