Skip to main content
. 2002 Apr;70(4):1684–1693. doi: 10.1128/IAI.70.4.1684-1693.2002

FIG. 2.

FIG. 2.

AM from HIV-positive individuals produce less MIP-1β than control AM in response to M. tuberculosis. Supernatants of AM from normal (n = 8) and HIV-infected (n = 8) individuals were infected with M. tuberculosis as described for Fig. 1 and assayed by ELISA for MIP-1β. Induction of MIP-1β in normal AM was comparable for both H37Ra and H37Rv. AM from the HIV group produced significantly less MIP-1β than did normal AM in response to both H37Ra and H37Rv. Asterisks indicate a significant difference compared to uninfected (Un) AM. Number signs indicate a significant difference compared to the corresponding condition for AM from HIV-infected individuals.