. 2000 Oct 5;67(5):1154–1162. doi: 10.1016/s0002-9297(07)62946-2

Table 4.

Regions Showing Evidence for Linkage to Asthma, Atopy, or Related Phenotypes among the Hutterites (Table 2) and in Other Genome Screens^[Note]

Chromosome	Distance from pter(cM)	Present Study	Busselton^a	United States^b	Germany^c	France^d
1	4–14	Asthma	…	…	Specific IgE (P = .0013)	…
	30–37	Asthma	…	…	Eosinophils (P = .009)	…
	139–150	+SPT	…	…	IgE, specific IgE (P = .0098, .0045)	…
2	186–215	+SPT	…	…	IgE (P = .0016)	…
5	33–52	BHR	…	…	Specific IgE (P = .0027)	…
9	95–112	Symptoms	…	…	Asthma (P = .0073)	…
11	21–43	+SPT	…	Asthma (LOD = 1.2)^e	Specific IgE (P = .0036)	IgE (P=.002)
16	105–125	+SPT	IgE (P < .01)	…	…	…
19	67–70	BHR	…	Asthma (LOD = 2.0)^e	…	BHR (P = .01)
20	32–39	+SPT	Wheeze (P < .01)	…	…	…

Note.— Only values P < .01 are shown. A review of all of the regions of overlap between the five genomewide screens is presented elsewhere (Ober and Moffatt 2000).

Source: Daniels et al. (1996).

Sources: The Collaborative Study on the Genetics of Asthma (1997); Mathias et al. (2000).

Sources: Wjst (1999); Wjst et al. (1999).

Source: Dizier et al. (2000).

In white families.