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. 2026 Mar 10;16:8699. doi: 10.1038/s41598-026-43501-z

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© The Author(s) 2026

Open Access This article is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License, which permits any non-commercial use, sharing, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if you modified the licensed material. You do not have permission under this licence to share adapted material derived from this article or parts of it. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc-nd/4.0/.

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Fig. 4 — IGF2BP1 had the potential to predict the sensitivity of therapeutic drugs for PTC. (A) PCA analysis of CTL cells. (B) Cell proportion distribution in different groups. (C) Utilize the Monocle 2 algorithm to analyze sequences and loci of 17 tumor cell subsets. (D) The correlations between IGF2BP1 and immune score. (E)The relation between IGF2BP1 and immune checkpoint-related genes.