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. 2026 Mar 30;35(2):57–80. doi: 10.5607/en25040

Table 4.

Small molecules enhancing Aβ clearance

Name (alternative name) Mechanism of action Clinical phase
(current status)
Pathological biomarker Cognition Ref
RAGE antagonists
Azeliragon (TPP488) RAGE antagonist Phase 3 (STEADFAST, terminated for futility) No data available No benefit overall; *HbA1c ≥6.5% ↑ (ADAS-Cog11) [118]
ApoE lipidation enhancers
Bexarotene RXR agonist Phase 2 (completed) Amyloid PET: No benefit overall; *APOE4 noncarrier ↓ (regional amyloid)
*Serum Aβ42 ↑ (correlated with decreased cortical amyloid in treated APOE4 noncarrier)
No benefit overall
*APOE4 noncarrier ↓ (MMSE)
[113, 126]
CS6253 ABCA1 modulator Phase 1 (completed) No data available No data available [127]
Microglia modulators
GV-971 Gut-brain axis immunomodulator Phase 3 (China, completed→ conditional approval in China)
Phase 3 (GREEN MEMORY, suspended for COVID-19)
No data available *↑ (ADAS-Cog12); *MMSE 11-14 ↑ (CIBIC+) [129]
VG-3927 TREM2 agonist Phase 1 (completed) No data available No data available [133, 134]
IDE enhancers
Nasal Insulin IDE-mediated Aβ clearance65 Phase 2/3 (SNIFF, completed) CSF Aβ42/Aβ40 and Aβ42/total tau ratios: No benefit overall; *Device 1 subgroup ↑ No benefit overall; *Device 1 subgroup ↑ (ADAS-Cog12, ADL-MCI) [139]

*Asterisks indicate statistically significant differences. Arrows indicate the direction of change: for cognitive outcomes, ↑ and ↓ represent improvement or worsening, respectively; for pathological biomarkers, ↑ and ↓ indicate numerical increase or decrease. (-) indicates no change.