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. Author manuscript; available in PMC: 2026 Jun 29.
Published before final editing as: J Allergy Clin Immunol. 2026 Apr 28:S0091-6749(26)00292-7. doi: 10.1016/j.jaci.2026.04.012

Table 2.

Detailed lung phenotypes of STAT3-HIES.

Variable (n, %) All, n = 164 DBD variants, n = 89 SH2 or TA domain variants, n = 75 R382 variants, n = 49 V637 variants, n = 26
Asthma 41 (25.0) 22 (24.7) 19 (25.3) 11 (22.4) 5 (19.2)
ABPA/ABPM 9 (5.5) 7 (7.9) 2 (2.7) 3 (6.1) 1 (3.8)
Tracheal diverticuli 39 (23.8) 18 (20.2) 21 (28.0) 10 (20.8) 8 (30.8)
Aspergillus infection 55 (33.5) 32 (36.0) 23 (30.7) 17 (34.7) 9 (34.6)
Pseudomonas infection 51 (31.1) 29 (32.6) 22 (29.3) 17 (34.7) 9 (34.6)
Non-tuberculous mycobacterial infection 19 (11.6) 10 (11.2) 9 (12.0) 4 (8.2) 2 (7.7)
Lung parenchyma surgery 42 (25.6) 19 (21.4) 23 (30.7) 10 (20.4) 9 (34.6)
Prolonged bronchopulmonary fistula (> 2 weeks) 20 (12.2) 14 (15.7) 6 (8.0) 9 (18.4) 2 (7.7)
Long-term oxygen supplementation 12 (7.3) 5 (5.6) 7 (9.3) 1 (2.0) 3 (11.5)
Non-invasive ventilation 12 (7.3) 4 (4.5) 9 (12.0) 2 (4.1) 2 (7.7)

Abbreviations: HIES, hyper-IgE syndrome; DBD, DNA-binding domain; SH2, Src homology 2; TA, transactivation; ABPA/ABPM, allergic bronchopulmonary aspergillosis/mycosis.