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. 2026 Jul 24;75(7):002189. doi: 10.1099/jmm.0.002189

Fig. 2. Antibiotic disc diffusion assays comparing A. baumannii AB5075 wild-type (WT) and Δp1AB5075 strains. MH2 agar plates were lawned with wild-type A. baumannii AB5075 and Δp1AB5075, and antibiotic-containing discs were placed on the agar surface. The plates were incubated for 24 h at 37 °C. (a) AK (amikacin 30 µg), SXT (trimethoprim–sulphamethoxazole 25 µg), TOB (tobramycin 10 µg), S (streptomycin 25 µg), CN (gentamicin 30 µg), K (kanamycin 5 µg), (b) nitrofurantoin (F 100 µg), D (doripenem 10 µg), MEM (meropenem 10 µg), VA (vancomycin 20 µg), IPM (imipenem 10 µg), E (erythromycin 15 µg), (c) TGC (tigecycline, 15 µg), TE (tetracycline, 30 µg), OX (oxacillin, 1 µg), FOX (cefoxitin, 30 µg), TZP (piperacillin/tazobactam, 110 µg), TIC (ticarcillin, 75 µg), (d) CAZ (ceftazidime, 30 µg), CFP (cefoperazone, 30 µg), FEP (cefepime, 30 µg), ATM (aztreonam, 30 µg), C (chloramphenicol, 30 µg) and CIP (ciprofloxacin, 5 µg).

Disc diffusion assay plates showing WT and delta p1AB5075 strains tested against aminoglycosides, carbapenems, tetracyclines, and cephalosporins. The mutant displays larger inhibition zones across most antibiotic classes.