Figure 8.

Hypothetical Model for Harpin_Psph-Induced Signal Transduction in Tobacco.

Activation by harpin_Psph of pathogen defense responses in tobacco may be mediated by receptor-mediated membrane insertion (1), by direct insertion of harpin_Psph into membranes, or by a specific (non)proteinaceous receptor (2). Alternatively, membrane insertion and receptor-mediated recognition of harpin_Psph may occur independently, with either or both pathways activating MAPK-dependent HIN1 expression in tobacco.