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. Author manuscript; available in PMC: 2006 Mar 8.
Published in final edited form as: Nat Neurosci. 2003 May;6(5):532–537. doi: 10.1038/nn1042

Fig. 1.

Fig. 1

Learning-related inhibition of complex spikes parallels the development of eyeblink conditioning. (a) Eyeblink CRs for P17 and P24 rats that received paired or unpaired CS–US presentations. Inset, left, slow and fast sweep (asterisk in slow sweep) of raw US-elicited complex spike activity from a Purkinje cell in an eye region of lobule HVI (arrows indicate complex spikes); scale bars = 105 ms, 55 μV; 18 ms, 40 μV. Inset, right, averaged integrated EMG activity from CR trials for the P17 (gray) and P24 (black) rats from which complex spikes in b–e were recorded. (b–e) Histograms (left) and rasters (right) of single complex spikes from a P17 rat on trials without (b) and with (c) CRs and a P24 rat on trials without (d) and with (e) CRs.