Table 3.
Type | Mechanism of action/principle | Wound type | Evidence; current status |
---|---|---|---|
Radiant heat dressing | Improves tissue oxygenation and increases subcutaneous oxygen tension | Mainly postoperative wounds; diabetic ulcers; pressure ulcers | Limited evidence; not in routine use |
Ultrasound therapy | Mechanical effect causing micromassage of tissue; anti-inflammatory effect (due to reduction in macrophages) | Pressure ulcers and VLU | Limited evidence; not in routine use |
Laser | Stimulates fibroblast activity and collagen metabolism; promotes neovascularisation; inhibits inflammation | VLU, diabetic ulcers, and burns | Limited evidence; not in routine use |
Hydrotherapy | Form of mechanical debridement; removes loosely attached devitalised tissue and other cellular debris from wound bed | Pressure ulcers, VLU, and other chronic wounds containing excess slough or necrotic tissue | Practised in USA, but not well established in UK |
Electrotherapy | Stimulates body's endogenous bioelectric system by delivering therapeutic levels of electric current into wound | TENS* is used to treat some ischaemic ulcers, diabetic foot ulcer, and pressure ulcers | Limited evidence; TENS* used in specialist centres |
Electromagnetic therapy | Promotes cytokine synthesis in the topically applied mononuclear cells (autologous) | Ischaemic ulcers, pressure ulcers, and VLU | Limited evidence; not in routine use |
VLU = venous leg ulcer.
Form of electrotherapy.