Fig. 3. Variegation of Wink-A7 responds to modifiers. Eye pigment measurements. Lane 1, Wink-A7 control male; lane 2, variegation caused by heat shock induction of GAL4–HP1; lane 3, partial suppression of variegation by a null mutation of Su(var)2-5 (Eissenberg et al., 1992); lane 4, an excess dose of HP1 does not enhance variegation of Wink-A7; the excess HP1 results from the heat-shock-inducible transgene in HSHP1.83C (Eissenberg et al., 1992); lane 5, partial suppression of variegation by the Su(var)3-7 mutation Df(3R)Ace^HD1; lane 6, an excess dose of Su(var)3-7 does not enhance variegation of Wink-A7; the excess Su(var)3-7 results from the heat-shock-inducible transgene HSFLTX (Cléard et al., 1995). Lanes 7 (control) and 8 (heat shock induction of GAL4–HP1), test of another insertion of Winkelried in euchromatin (Wink-S located at 43A). Variegation is not induced.