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. 2005 Nov;171(3):1057–1081. doi: 10.1534/genetics.104.038018

TABLE 1.

GMR-dRetMEN2 genetically interacts the Ras, Src, and JNK pathways

Gene Gene function Allele GMR-dRetWT GMR-dRetMEN2A GMR-dRetMEN2B
spitz dEGF receptor ligand spitzs3547 WS(65) WS(86)
spitz01068 WS(46)
spitz1 WS(40) WS(56)
drk Ortholog of Grb2 protein drkk02401 N N SS(95)
drkk13809 WS(23) N WS(100)
drk10626 MS(100) WS(24) SS(100)
Sos RasGTP-exchange factor Sos34Ea-6 SS(100)
Gap1 RasGTPase activating factor Gap1B2 ME(86) SE(100) SE(97)
Ras85D Ras ortholog Ras85D06677 MS(100) WS(76)
Ras85DΔc40b WS(67)
ksr Kinase suppressor of Ras ksrS-627 N MS(100)
ksrj5E2 WS(18) WS(84) WS(67)
phl Raf kinase phl11.29 N
phlPB26 N
Dsor MAP kinase kinase Dsor1 N
Src64B Src kinase ortholog Src64BP1 WS(86) WS(65)
Src42A Src kinase ortholog Src42Ak10108 WS(50) WS(74)
bsk c-jun kinase (JNK) bsk1 WS(47) WS(44)
bsk2 WS(43) WS(86)
Jra c-jun transcription factor JraIA109 WS(46) WS(56) WS(61)
dock Ortholog of Nck adaptor dockk13421 N
dock04723 N
dsh Wnt, JNK signaling dsh3 N N
dsh6 N N N

Genetic interactions are indicated according to strength of the phenotype: W, weak; M, moderate; and S, strong; and the type of interaction: S, suppressor; E, enhancer; N, no interaction. WE, for example, indicates a weak enhancer. Parentheses indicate penetrance of the interaction as a percentage.