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. 2001 Jan 16;98(2):676–682. doi: 10.1073/pnas.98.2.676

Table 1.

Data obtained from subjects in group I while they rested quietly but awake with their eyes closed

Area x y z OEF P CBF P CMRO2 P
A M 31/7 −5 −49 40 1.010 0.62 1.374* <0.0001 1.397* <0.0001
B L 40 −53 −39 42 0.901 0.07 0.735* <0.0001 0.695* <0.0001
C L 39/19 −45 −67 36 0.987 0.66 0.813* 0.0008 0.805* 0.0003
D R 40 45 −57 34 0.978 0.26 1.002 0.96 0.998 0.97
E L lateral 8 −27 27 40 0.981 0.52 0.991 0.74 0.986 0.74
F L 8/9 −11 41 42 0.902 0.04 0.874 0.006 0.803* 0.001
G R 8/9 5 49 36 0.887 0.004 0.893 0.01 0.813* 0.001
H L 9 −15 55 26 0.943 0.35 0.813* 0.0001 0.785* 0.002
I L 10 −19 57 8 0.971 0.06 0.940 0.01 0.927 0.01
J M 10 −1 47 −4 0.933 0.01 1.284* <0.0001 1.203* <0.0001
K L 10/47 −33 45 −6 0.930 0.25 0.920 0.03 0.879 0.04
L M 32 3 31 −10 0.946 0.20 1.111 0.005 1.058 0.26
M L 20 −49 −19 −18 0.972 0.46 0.821* <0.0001 0.814* 0.0002

The values for OEF, CBF, and CMRO2 are expressed as local-to-global ratios (see Methods). For the 19 subjects in group I the global values for OEF and CBF (± SD) were 0.40 ± 0.09 (dimensionless) and 46 ± 8 ml/(min × 100 g), respectively. The mean arterial oxygen content for this group was 16.6 ± 0.16 ml/ml. From these data quantitative images of the CMRO2 were created that yielded a mean cerebral hemisphere value of 2.94 ± 0.41 ml/(min × 100 g) or 1.31 ± 0.18 μmol/(min × g). The asterisks denote values that differ significantly from the global mean after correction for multiple comparisons.