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Intravenous coadministration of native IFN-α2 and FMS–IFN-α2 facilitates prolonged circulating antiviral activity from the time of administration. Groups of mice (n = 3 for each group) received intravenously native IFN-α2 (10 μg/mouse) or FMS7–IFN-α2 (10 μg/mouse) or both (10 μg of each). Blood aliquots were withdrawn at the indicated time points and analyzed in human WISH cells for antiviral activity.
