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β-CCE enhances ethanol effects and is an agonist on α4β3δ receptors. (a) The β-carboline β-CCE at the indicated concentrations was applied alone or together with ethanol (always in the presence of 300 nM GABA) to oocytes expressing α4β3δ receptors, and peak GABA/Cl− currents were measured. (b) Dose-dependent reversal of 300 nM β-CCE enhancement of α4β3δ currents by Ro15-4513.
