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. 2003 May;41(5):1929–1935. doi: 10.1128/JCM.41.5.1929-1935.2003

TABLE 5.

Distribution of genes encoding VFs among RAPD and ECOR groupsa

VF gene No. of genes in ECOR groupb:
Pc No. of genes in RAPD groupb:
Pd No. of genes in other strainsb (n = 8)
A (n = 12) B1 (n = 7) B2 (n = 71) D (n = 15) I (n = 61) II (n = 36)
neuC 4 (33) 2 (29) 46 (65) 6 (40) 0.0045 37 (61) 14 (39) 0.04 7 (87)
hly 0 0 38 (53) 0 <0.0001 27 (44) 8 (22) 0.03 3 (37)
fimH 7 (58) 6 (86) 60 (84) 11 (73) NSe 54 (88) 23 (64) 0.004 7 (87)
iucC 5 (42) 2 (29) 43 (61) 9 (60) NS 41 (67) 14 (39) 0.007 4 (50)
papC 1 (8) 0 19 (27) 2 (13) 0.033 16 (26) 3 (8) 0.03 3 (37)
afa 2 (17) 0 1 (1) 0 NS 0 2 (6) NS 1 (12)
sfa/foc 1 (8) 0 34 (48) 0 <0.0001 26 (43) 7 (19) 0.018 2 (25)
ibeA 1 (8) 0 15 (21) 1 (7) 0.046 15 (25) 1 (3) 0.005 1 (12)
Median (range) 2 (0-4) 1 (0-3) 4 (1-6) 2 (0-4) 4 (0-6) 2 (0-4) 3.5 (2-6)
a

K1 capsule antigen (neuC), alpha-hemolysin (hly), type 1 pili (fimH), aerobactin (iucC), type P pili (papC), afimbrial adhesin (afa), type S pili and F1C fimbriae (sfa/foc), and IbeA protein (ibeA).

b

Number (percent) of strains positive for the virulence gene as demonstrated by a PCR assay.

c

ECOR group B2 versus all other ECOR groups.

d

RAPD group I versus RAPD group II.

e

NS, not significant.