Table 4.

Logistic regression models testing effects of prenatal PAH exposure on the odds of mental and psychomotor development delay at 12, 24, and 36 months^a (n = 181).

	Model 1: 12 months			Model 2: 24 months			Model 3: 36 months
	β	p-Value	Exp(β)	β	p-Value	Exp(β)	β	p-Value	Exp(β)
Dependent variable: moderate delay (MDI < 85)
Constant	2.65	0.67	14.12	3.04	0.53	20.92	6.68	0.24	798.33
PAHs	−0.19	0.71	0.82	−0.16	0.68	0.86	1.06	0.01	2.89
Ethnicity (1 = African American; 0 = others)	0.32	0.50	1.37	−0.90	0.01	0.41	−0.77	0.06	0.46
Sex (1 = male)	0.66	0.12	1.94	0.84	0.01	2.31	0.50	0.16	1.65
Gestational age	−0.07	0.67	0.93	−0.02	0.89	0.98	−0.10	0.47	0.90
Home environment	−0.05	0.19	0.95	−0.06	0.03	0.94	−0.10	< 0.01	0.90
Dependent variable: moderate delay (PDI < 85)
Constant	−1.50	0.82	0.22	−1.11	0.87	0.33	−2.30	0.79	0.10
PAHs	−0.92	0.16	0.40	0.41	0.41	1.51	−0.22	0.72	0.80
Ethnicity (1 = African American; 0 = others)	0.39	0.41	1.48	−0.21	0.67	0.81	−0.65	0.28	0.52
Sex (1 = male)	−0.18	0.68	0.83	−0.42	0.35	0.66	0.01	0.98	1.01
Gestational age	−0.01	0.97	0.99	−0.02	0.89	0.98	0.04	0.85	1.04
Home environment	−0.01	0.88	0.99	0.01	0.86	1.01	−0.05	0.25	0.95

^aModels were also adjusted for prenatal ETS and CPF. Further inclusion of maternal IQ and maternal education as covariates did not alter the results.