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. 2002 Feb 15;16(4):490–502. doi: 10.1101/gad.218202

Figure 4.

Figure 4

Epistatic analyses. (A) Phenotypes of mock- (left) and BTH- (middle) treated acd11/nahG, acd11/pad4-2, and acd11/eds1-2 plants 6 d after treatment. Trypan blue staining of BTH-treated leaves is also shown (right). (B) Accumulation of PR1 and SAG13 mRNA in mock- and BTH-treated Ler, acd11/nahG, acd11/pad4-2 and acd11/eds1-2 plants 6 d after treatment. At longer exposures, weak PR1 and SAG13 signals are detected in BTH-treated acd11/eds1-2. (C) Phenotypes of acd11/npr1-1, acd11 and npr1-1 mutants. (D) Accumulation of PR1 and SAG13 mRNA in acd11/npr1-1, acd11 and npr1-1 mutants. (E) Accumulation of PR1 and SAG13 in acd11/ein2-1, acd11/jar1-1 and in single mutants. (F) Representative mock- or BTH-treated leaves of nahG and acd11/nahG after 4 d in light or dark.