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The angiotensin II (AngII, 100 nM) induced endothelial superoxide formation was abolished in the presence of the tyrosine kinase inhibitor genistein (30 μM, n=11, *P<0.05 vs control, #P<0.05 vs angiotensin II) while the protein kinase C inhibitors staurosporine and chelerythrine chloride (10 μM, see text) were not effective.
