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. 2003 May 29;139(2):399–407. doi: 10.1038/sj.bjp.0705260

Table 2.

Carbachol EC50 (M) in amylase release and IP production in the absence and the presence of the antagonists of the M2, M4 and nicotinic receptors

Additions Control Castrated
  Amylase IP Amylase IP
None 1.54 × 10−5 1.78 × 10−5 9.88 × 10−7 7.94 × 10−7
AF-DX 116 1.58 × 10−5 2.19 × 10−5 7.40 × 10−7 7.20 × 10−7
Tropicamide 1.41 × 10−5 1.66 × 10−5 7.90 × 10−7 8.10 × 10−7
Benzoquinonium 1.35 × 10−5 2.00 × 10−5 8.50 × 10−7 7.94 × 10−7

Parotid slices were preincubated in the absence or presence of 1 × 10−5 M of the antagonists added at the beginning of the incubation time, followed by determination of carbachol dose-dependent stimulation of amylase release or IP production as described in Methods. Results shown are one of four experiments with similar results.