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. 2003 Dec 22;141(2):322–328. doi: 10.1038/sj.bjp.0705603

Table 1.

Effect of N-nitro-L-arginine, haemoglobin and indomethacin on acetylcholine-induced relaxation of aortae from vehicle, daidzein- and 17β-oestradiol-treated rats

  Vehicle Daidzein 17β-oestradiol
Ach n pEC50 Rmax (%) n pEC50 Rmax (%) n pEC50 Rmax (%)
Control 6 7.10±0.08 89±4 6 7.32±0.1* 99±1* 6 7.34±0.04* 96±2*
L-NOARG 6 ND 0# 6 5.82±0.4# 28±6#* 6 5.93±0.19# 33±5#*
L-NOARG+Hb 6 ND 0# 6 6.33±0.3# 29±4#* 6 6.16±0.26# 28±3#*
Indomethacin 6 7.12±0.10 87±4 6 7.26±0.09 95±2* 6 7.25±0.12 94±3*
Indomethacin+L-NOARG 6 ND 0# 6 6.55±0.09# 21±10#* 6 6.64±0.31# 29±4#*

A comparison of the sensitivity (pEC50) and maximum relaxation to ACh in the absence (control) and presence of N-nitro-L-arginine (L-NOARG, 100 μM) alone, or with haemoglobin (Hb, 20 μM) in endothelium intact thoracic aortae from vehicle, daidzein- (0.2 mg kg−1 per day s.c.) and 17β-oestradiol- (0.1 mg kg−1 per day s.c.) treated rats. n=the number of experiments.

*

Significantly different from control ACh response in aortic rings from vehicle-treated rats (Bonferroni test, P<0.05).

#

Significantly different from the corresponding control ACh response within the same treatment group (Bonferroni test, P<0.05). ND=could not be calculated.