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. 2002 Jun;129(2):774–785. doi: 10.1104/pp.010969

Figure 1.

Figure 1

Inhibition of hypocotyl elongation in Arabidopsis seedlings. A, Hypocotyl lengths of seedlings germinated as described in “Materials and Methods” and maintained for the indicated lengths of time in darkness. Error bars represent the se. Wt, Wild type; Oe, CRY1 overexpressor; C1C2, cry1cry2 double-mutant seedlings. B, Seedlings were plated and placed under monochromatic light sources as indicated in “Materials and Methods.” Measurements of seedling hypocotyl growth are presented as percentages of dark-grown control seedlings (where there is no growth inhibition). Error bars represent the se. OE, Transgenic seedlings overexpressing CRY1 protein; Wt, wild-type seedlings; C1C2, cry1cry2 double-mutant seedlings. C, Plot of cryptochrome action spectrum for 20% and 30% hypocotyl growth inhibition, respectively, as calculated from the data used in B. Oe, Overexpressor of CRY1; Wt, wild type. Peaks represent peak activity (maximal sensitivity) of the photoreceptor.