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Effects of the PAR-1 agonists on gastrointestinal transit in mice. The human PAR-1-derived peptide SFLLR-NH2 (SFp-NH2) or the control peptide FSLLR-NH2 (FSp-NH2) (a) and the specific PAR-1 agonist TFLLR-NH2 (TFp-NH2) (b), in combination with i.p. amastatin at 2.5 μmol kg−1, were administered i.p. to the mouse. Data represent the mean±s.e.mean from 8–10 (a) or 4–6 (b) mice.
